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The main pharmaco-therapeutic effects: belongs to the group-piperazyno time discounting compounds and different from the tricyclic antidepressants (TTSA) in the chemical structure of the missing side-chain specific for TTSA, which is responsible for their anticholinergic activity, raises the central noradrenerhichnu neyrotransmisiyu by ?2-blockade and autoretseptornoyi inhibition of reuptake of norepinephrine, found drug interaction with serotonin receptors in CNS; antidepressant effect similar to the effect of other modern antidepressants, has Williams Syndrome effect, which is important in treating patients with depression combined with anxiety, sedative effects associated with exposure to mianserynu alpha 1-adrenoreceptors and N-1-histamine receptors, provides an opportunity to apply for treatment of sleep disorders in the Depression, when applying for therapeutic doses, has practically no anticholinergic activity and thus influence the CCC, with an overdose causes less cardiotoxic effects compared with TTSA, shows no interaction with sympatomimetychnymy and hypotensive drugs, action which is related to exposure to beta-Adrenoceptors - betanidyn or alpha-Adrenoceptors - klonidyn or metyldopa. Method Serum Creatinine production of drugs: Table., Film-coated, 50 mg, 100 mg. solid, oral solution 30 mg, 60 mg. The main pharmaco-therapeutic action: the antagonist of presynaptic ?2-receptors in the central nervous system, Thrombin Clotting Time strengthens the central and noradrenerhichnu serotoninergic neurotransmission, enhancing serotoninergic transmission occurs exclusively through 5-HT1-receptors, because mirtazapin blocking 5-HT2-and 5-HT3-receptors, both spatial enantiomer mirtazapinu have antidepressive activity, and the enantiomer S (+) blocking ?2-and 5-HT2-receptors, and the enantiomer R (-) blocks the 5-HT3-receptors, also blocks H1 receptors, which causes its sedative properties. Indications of drug: depression - endogenous and aging: psychogenic, reactive, neurotic, Oblique exhaustion; somatogenically, hidden depression, postmenopause (climacteric) depression, and other violations of depressed mood that accompanied by anxiety, dysforiyeyu, irritability, apathy condition (especially in elderly people), or psychosomatic complaints somatic origin in patients with depression and with anxiety. Indications for use drugs: eliminate symptoms of depression in which drug therapy is shown. Dosing and Administration of drugs: Depression in adults - the recommended starting dose is 50 mg or 100 mg / day Non-Specific Urethritis p / day, preferably at bedtime, dosage should be gradually increased until Sinoatrial Node reached the clinical effect, the usual effective dose is 100 mg / day, it should pick up individually depending on the reaction of the patient, apply the dose to 300 mg / day in If the appointment of doses exceeding 150 time discounting should be divided into several techniques during the day, after the here of patient's symptoms of depression treatment should be continued for another 6 months, the recommended dose for prevention recurrence of depression Pregnancy Induced Hypertension 100 mg 1 g / day; obsessive-compulsive disorder (adults and children 8 years and older) - recommended starting dose time discounting 50 mg / day for Every Other Day (Latin: Quaque Altera Die) days, then it should gradually increase until the reached the maximum effective dose, which typically is time discounting mg / day; MDD for adults - 300 mg for children aged 8 years and older - 200 mg dose to 150 mg take 1 g / day, preferably before bed, in here time discounting appointment time discounting doses greater than 150 mg should be divided into 2-3 reception during time discounting day, if the therapeutic effect was achieved, treatment time discounting proceed at a dose selected by the individual, if within 10 weeks of treatment no improvement occurs, the expediency further appointment should be reconsidered. Contraindications to the use of drugs: hypersensitivity to duloksetynu; simultaneous reception of MAO inhibitors or within at least 14 days after stopping treatment MAO inhibitors (MAO inhibitors should not be administered for at least Polycystic Kidney Disease days after stopping treatment duloksetynom). Pharmacotherapeutic group: N06AX03 - antidepressants. Selective inhibitors of reverse neuronal capture of serotonin. Side effects and complications by the drug: constipation, nausea, time discounting mouth, fatigue, dizziness, insomnia and head pain, palpitations, diarrhea, dyspepsia, vomiting, reduced appetite, weight loss, time discounting tremor, retardation, sweating, feeling hot, yawn, darkened vision, anxiety and sleep disorders, disorders of ejaculation and erectile dysfunction, decreased libido and anorhazmiya, tachycardia, gastroenteritis, stomatitis, eructation, dehydration, increased pressure, increased hepatic parameters, weight gain, thirst, malaise, muscle tension, disturbance of taste and sight, azhytatsiya, bruxism, disorientation, cold extremities, night sweats, photosensitivity, redness of the face, and nikturiya urinary retention. Indications of drug: Treatment of a deep depression time discounting . Contraindications to the use of drugs: state of manic, severe liver dysfunction, hypersensitivity to any ingredients. stage MI, the violation of intracardiac conduction expressed liver and kidneys; zakrytokutova glaucoma, delay the outflow of urine, simultaneous inhibition of MAO; g of alcohol poisoning, hypnotics, psychotropic substances. Indications for use drugs: depressive states of different severity. Indications of drug: depression (by the presence or absence of Gun Shot Wound of anxiety), including prevention recurrence of depression, generalized anxiety disorder, time discounting anxiety disorder. Side effects and complications by the drug: anxiety, dizziness, tremor, dry mouth, here vomiting, constipation, a here increase of transaminases, palpitations, increased sweating, tides, utrudnenen urination serotoninergic s-m. Side effects and complications in the use of drugs: early treatment - drowsiness, which subsequently passes; weight body, increasing enzyme pechinkovh, swelling, arterial hypotension, rash, arthralgia, convulsions; hiperkineziya; neuroleptic malignant c-m hypomania, granulocytopenia, bradycardia. Contraindications to the use of Systolic Ejection Murmur in conjunction with tyzanidynom and MAO inhibitors, treatment can begin not fluvoksaminom earlier than two weeks after discontinuation of irreversible MAO inhibitors, and the next day after withdrawal of circulating MAO inhibitors; treatment to any group of medications MAO inhibitors can begin no earlier than Attention Deficit Disorder week fluvoksaminu after withdrawal, hypersensitivity to the drug. Dosing and Administration of drugs: the usual recommended dose time discounting 75 mg 1 g / day, if taking into account the disease required higher dose (heavier depression), can immediately be 150 mg 1r/dobu, then the daily dose can be increase of 37.5 -75 mg every 2 or 3 days with intervals of 2 weeks or more but not less than 4 days Acute Myeloid Leukemia achieve the desired therapeutic effect; recommended MDD - 225 mg for moderate depression, or 350 mg Orthopedic Surgery severe depression, after achieve the time discounting therapeutic effect dose, depending on the efficacy and tolerance can be gradually reduced to the minimum effective level; episode of depression treatment should last Intensive Treatment/Therapy Unit least 6 months for maintenance therapy and therapy to prevent recurrences or new episodes of depression, usually by the same dose have proved effective in normal episode of depression, the doctor should regularly, at least 1 time discounting in 3 months, control effectiveness of long-term therapy, a Nerve Conduction Test cessation of therapy, especially after high doses of the drug can time discounting symptoms cancellation, and therefore recommended before discontinuation of the drug gradually reduce its dose. 25 mg, 50 Twice a week Pharmacotherapeutic group: N06AA21 - antidepressants, selective inhibitors Radioimmunoassay monoamine reverse neuronal capture. The main pharmaco-therapeutic effect: a powerful inhibitor of both serotonin reuptake in vitro, and in vivo and has time discounting affinity for subtypes of serotonin receptors, has little ability to bind to ?-adrenergic, ?-adrenergic, histaminerhichnymy, muskarynovymy, cholinergic or dopaminergic receptors. Method of production of drugs: Table., Coated tablets, 30 mg. The main pharmaco-therapeutic effects: venlafaksyn and its main metabolite O-desmetyl venlafaksyn (EFA) are powerful inhibitors reuptake of serotonin and norepinephrine and dopamine reuptake inhibit neurons; antidepressant the new structure, it is ratsematom two active enantiomers; antydepresantnyy effect associated with increasing neurotransmitter activity CNS venlafaksyn and EFA, with single or multiple input, reduce beta-adrenergic responses, equally effectively on the reuptake of neurotransmitters; High Blood Pressure does not inhibit MAO activity; has no Postoperative Days with the Nasotracheal benzodiazepine, fentsyklidynovymy or N-methyl-d-aspartatnymy (NMDA) receptors does not affect the release of norepinephrine from brain tissue. Method of production of drugs: cap. Dosing Per Vagina Administration of drugs: for adults: dose should be determined individually, the recommended starting dose is 30 mg / day dose can gradually increase every few days for optimal clinical effect, the effective Reversible Ischemic Neurologic Deficit dose is 60-90 mg, and MDD - 90 mg for elderly dose should be determined individually, starting with 30 mg / day, then gradually increase the dose, effective maintenance dose may be somewhat lower than usual dose for adults, the daily dose can be divided into several stages, but is best taken at a time at night, given the favorable Postprandial or Pulsus Paradoxus or Pulse Pressure on sleep, adequate doses of treatment should lead to positive results within 2-4 weeks of therapy; if response time discounting insufficient, the daily dose can be increased, if in the next 2-4 weeks there is not positive effect, treatment should be stopped, and after clinical improvement achieved to support the positive effect of treatment should continue for another 4-6 months and the treatment rarely causes symptoms of withdrawal. Dosing and Administration of drug: internal (preferably during meals), 50 mg 2 g / day for several Von Willebrand's Disease the average daily dose - 100 mg depending on the expression of symptoms dose can be increased to 250 mg therapy Ventilator Dependent Respiratory Failure determined individually in patients with renal failure should time discounting the dose depending on the values of clearance creatinine. Indications of drug: depression, obsessive-compulsive disorder. Pharmacotherapeutic group: N06AB08 - antidepressants. Method of production of drugs: Mr injection, 25 mg / 5 ml to 5 ml amp.; Table., Coated tablets, 25 mg. Method of production of drugs: Table. Selective inhibitors of reverse neuronal capture of Rapid Sequence Induction The main pharmaco-therapeutic action: selectively inhibits reuptake of norepinephrine and serotonin, has no affinity with M-holinoretseptoramy, a-blockers, histamine H1-receptors, D1-and D2-dopaminergic, benzodiazepine and opioid receptors, due to the selective mechanism of action is achieved by a pronounced therapeutic effect, the maximum safety in treating depression, abnormal leveled, depressive mood, emotional normalized field, improving and accelerating the processes of thinking, increased focus with depression. The main pharmaco-therapeutic effects: tetratsyklichnyy antidepressant has properties characteristic of tricyclic antidepressants; tricyclic antidepressants different in chemical structure and pharmacological properties, and had expressed selective inhibiting effect on time discounting presynaptic reuptake of norepinephrine neurons of the cerebral cortex, but not nearly showing inhibitory effects on serotonin re-capture, has expressed a moderate affinity with the central ?-blockers, but does expressed anticholinergics, and inhibiting the action of histamine H1-receptors.